Tobacco use and lung cancer essay

Quitting smoking lowers the risks for cancers of the lung, mouth, throat, esophagus, and larynx. Screening for cervical and colorectal cancers helps find these diseases at an early, and often highly treatable, stage.

Jesse Steinfeld had raised concerns about this topic, leading to its inclusion in that report. For carbon monoxide CO specifically, levels in enclosed spaces could exceed levels then permitted in outdoor air. The possibility that CO emitted from cigarettes could harm persons with chronic heart or lung disease was also mentioned.

The report covered exposures and potential health consequences of involuntary smoking, and the researchers concluded that smoking on buses and airplanes was annoying to nonsmokers and that involuntary smoking had potentially adverse consequences for persons with heart and lung diseases. Two studies on nicotine concentrations in nonsmokers raised concerns about nicotine as a contributing factor to atherosclerotic cardiovascular disease in nonsmokers.

Tobacco use and lung cancer essay chapter concluded with recommendations for research including epidemiologic and clinical studies. That chapter commented on the methodologic difficulties inherent in such studies, including exposure assessment, the lengthy interval during which exposures are likely to be relevant, and accounting for exposures to other carcinogens.

Involuntary smoking was also reviewed in the report, which focused on chronic obstructive pulmonary disease and smoking USDHHS Chapter 7 Passive Smoking of that report included a comprehensive review of the mounting information on smoking by parents and the effects on respiratory health of their children, data on irritation of the eye, and the more limited evidence on pulmonary effects of involuntary smoking on adults.

The chapter began with a compilation of measurements of tobacco smoke components in various indoor environments. The extent of the data had increased substantially since Bythe data included measurements of more specific indicators such as acrolein and nicotine, and less specific indicators such as particulate matter PMnitrogen oxides, and CO.

The report reviewed new evidence on exposures of nonsmokers using bio-markers, with substantial information on levels of cotinine, a major nicotine metabolite. The report anticipated future conclusions with regard to respiratory effects of parental smoking on child respiratory health Table 1.

In its pages, the report covered the full breadth of the topic, addressing toxicology and dosimetry of tobacco smoke; the relevant evidence on active smoking; patterns of exposure of nonsmokers to tobacco smoke; the epidemiologic evidence on involuntary smoking and disease risks for infants, children, and adults; and policies to control involuntary exposure to tobacco smoke.

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That report concluded that involuntary smoking caused lung cancer in lifetime nonsmoking adults and was associated with adverse effects on respiratory health in children.

The report also stated that simply separating smokers and nonsmokers within the same airspace reduced but did not eliminate exposure to secondhand smoke. All of these findings are relevant to public health and public policy Table 1. The lung cancer conclusion was based on extensive information already available on the carcinogenicity of active smoking, the qualitative similarities between secondhand and mainstream smoke, the uptake of tobacco smoke components by nonsmokers, and the epidemiologic data on involuntary smoking.

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The three major conclusions of the report Table 1. In its monograph on tobacco smoking, the agency supported this conclusion on the basis of the characteristics of sidestream and mainstream smoke, the absorption of tobacco smoke materials during an involuntary exposure, and the nature of dose-response relationships for carcinogenesis.

In reaching this conclusion, the NRC report cited the biologic plausibility of the association between exposure to secondhand smoke and lung cancer and the supporting epidemiologic evidence. On the basis of a pooled analysis of the epidemiologic data adjusted for bias, the report concluded that the best estimate for the excess risk of lung cancer in nonsmokers married to smokers was 25 percent, compared with nonsmokers married to nonsmokers.

With regard to the effects of involuntary smoking on children, the NRC report commented on the literature linking secondhand smoke exposures from parental smoking to increased risks for respiratory symptoms and infections and to a slightly diminished rate of lung growth.

Sincethe conclusions with regard to both the carcinogenicity of secondhand smoke and the adverse effects of parental smoking on the health of children have been echoed and expanded Table 1. Inthe U. A comprehensive meta-analysis of the 31 epidemiologic studies of secondhand smoke and lung cancer published up to that time was central to the decision to classify secondhand smoke as a group A carcinogen—namely, a known human carcinogen.

Estimates of approximately 3, U. The report also covered other respiratory health effects in children and adults and concluded that involuntary smoking is causally associated with several adverse respiratory effects in children. There was also a quantitative risk assessment for the impact of involuntary smoking on childhood asthma and lower respiratory tract infections in young children.

Surgeon General, addressing adverse effects from exposure to tobacco smoke In the decade since the EPA report, scientific panels continued to evaluate the mounting evidence linking involuntary smoking to adverse health effects Table 1. The epidemiologic evidence on involuntary smoking has markedly expanded sinceas have the data on exposure to tobacco smoke in the many environments where people spend time.Since this is a deductive essay, one is supposed to give several points that can be used to lead to a specific conclusion which, in this case, is that tobacco smoking is a deadly habit.

Get information on cigarette, cigar, and smokeless tobacco use, and learn how it affects different groups of people. Read more. How Tobacco and Smoking Affects Your Health. Harmful Chemicals in Tobacco Products; Lung Cancer; Prostate Cancer; View All Cancer Types; Back To Top. Imagine a world free from cancer. Help make it a reality. DONATE. The incidence of lung cancer is strongly correlated with cigarette smoking, with about 90% of lung cancers arising as a result of tobacco use. The risk of lung cancer increases with the number of. Lung cancer is known to be fatal and causes death after if not treated immediately the symptoms manifest. A persuasive essay about smoking can help smokers understand the risks that are associated with the act of smoking. For instance, when a parent smokes next to his children it can hurt in several ways. Smoking tobacco causes cancer essay.

It is a good practice that one should make known what is to be deduced from the content of the essay early enough in the essay so a reader can tell what the point of the essay is. - Lung Cancer Lung cancer is the leading cancer killer among both men and women.

There are two different types of lung cancer: nonsmall cell lung cancer and small cell lung cancer. There are also many different things that can cause lung cancer, but the number one cause for lung cancer is tobacco use. Tobacco Use and Lung Cancer Essay - Lung Cancer Lung cancer is the leading cancer killer among both men and women.

There are two different types of lung cancer: nonsmall cell lung cancer and small cell lung cancer. Tobacco use has predominantly negative effects on human health and concern about health effects of tobacco has a long history.

a metaanalysis citing other works to link tobacco use to lung cancer. Lickint showed that lung cancer sufferers were likely to be smokers. A few dangerous diseases one could get by using tobacco include: coronary heart disease, lung, oral, throat, kidney, and pancreatic cancer, and even COPD.

Since the publication of that report, more than 20 million Americans have died because of smoking. Tobacco use causes: Cancer (oropharynx, larynx, esophagus, trachea, bronchus, lung, acute myeloid leukemia, stomach, liver, pancreas, kidney and ureter, cervix, .